——在破败的DMars办公大楼里高层资料柜找到的一张报告
We have already taken part of the 'Black Window' project.
We did some secret experiments on this it.
The first is the confirmation of information about the FLo Research Institute， and the result is that the information is basically consistent.
We extracted the virus from the abnormal tissues and obtained a small amount of the original virus. These viruses will be used to study structural features.
Because of their abnormal metabolism and activity， infected cells have an unusual ability to replicate. According to statistics， under ideal conditions， the infected tissue can acquire adherent wall within 1-2 hours， and contact inhibition will occur after 1-2 days of growth.
We don't know for sure why virus-infected cells grow instead of lysing. And what we've found is that when a virus infects a cell， it seems to be lurking somewhere in the cell or tissue， and basically doesn't replicate itself. Sometimes the virus is activated and the acute attack causes the infected cell to lyse and produce a large number of viruses.
At present， it is not clear what is the cause of latent virus activation， but from the basic data， the virus almost appears latent state after infecting cells， which means that the re-infectivity is very weak.
Our guess for abnormal tissue behavior is that the latent virus modifies the genes of the infected cells or is treated by the cell transcriptase as part of the genes. So they show abnormal growth behavior and metabolism.
For the infected cells that can still divide normally and show no special characteristics， we developed two experimental protocols.
Experiment 1: If nutrients continued to be supplied， we found that the contact inhibition of the subjects began to collapse， and the tissue cells continued to grow until they grew and piled up in three dimensions. But as more and more cells are produced， the waste organic matter causes the pH of the petri dish to change and the cells begin to die away.
As we clean up the waste， the cells continue to divide， and as they accumulate more and more， the waste becomes more and more difficult to clean up. Tissue cells begin to damage again.
Experiment 2: If there is no continuous supply of nutrients， the subjects will divide slowly， most of them reach the saturated density and a few of them will no longer divide without reaching the saturation density. At this time， the number of cells is almost unchanged. But not for a few hours， the petri dishes were detected to excrete large amounts of organic matter. In order not to disturb the subjects， we cleaned up， but the cells still died too fast， and eventually the cells lysed gradually， producing a large number of viruses.
The results of two experiments showed that the infected tissue cells could still maintain normal cell division， but the ability to break through contact inhibition showed certain characteristics of cancer cells.
This discovery， we hope， will help us to study cancer cells in general， and to attack them， and perhaps also to help human diseases.
Infected cells metabolize faster than normal cells， and we're going to look more closely at the internal structure of infected cells.
We still need to know more about the characteristics of the virus and will continue to study further.
This particular virus we temporarily replaced the number R1 virus.
S report，21st Aug，2020
——
（后人翻译中文如下：）
黑窗计划内容我们已经拿了一部分。
关于这些，我们进行了一些秘密实验。
首先是关于FLo研究所的信息确认，结果是信息基本吻合。
我们将异常组织进行病毒提取，得到了少量原病毒。这些病毒将会用在研究结构特征上。
因为其异常代谢缘故和活跃程度，感染细胞具有不同寻常的复制能力。据统计，在理想条件下，感染组织能在1-2小时即获得贴壁，生长至1-2天后便发生接触抑制。
我们尚不明确为什么病毒感染细胞能不会裂解反而能生长。我们研究发现病毒感染细胞后会似乎潜伏者细胞或组织某处，基本不进行复制本身。有时病毒被激活从而急性发作使感染细胞裂解产生大量病毒。
目前也尚不明确何种原因是潜伏病毒激活，但从基本数据来看，病毒感染细胞后几乎呈潜伏状态，意味着再感染力很弱。
我们对于异常组织表现猜测为：潜伏病毒对感染细胞的基因进行了修改或者被细胞转录酶当做基因的一部分。所以表现出异常生长行为和代谢。
对于感染细胞仍能进行正常分裂却不表现特殊特征，我们制定了两个实验方案。
实验一：若是继续供给营养物质，我们则发现实验对象的接触抑制开始崩塌，组织细胞继续生长，直至向三维维度生长并堆积。但是由于细胞越来越多，产生废弃物有机物导致培养皿pH值变化使细胞开始逐渐死亡。
我们将废弃物清理，细胞仍能继续进行分裂，细胞堆积越来越多，废弃物开始越来越难清理。组织细胞再次开始受损。
实验二：若不继续供给营养物质，实验对象缓慢分裂，大部分到达饱和密度少部分没有达到饱和密度就不再分裂了，此时细胞数量几乎不变。但维持不了几个小时，检测到培养皿出现大量排泄有机物。为了不干扰实验对象我们进行了清理，但是细胞的死亡速度仍然过快，最后细胞逐渐裂解，产生大量病毒。
两次实验结果，得出证明，此被感染的组织细胞仍能保持细胞正常分裂，但是能够突破接触抑制却表现了一定的癌细胞特征。此发现我们希望有助于我们研究寻常癌细胞，并加以攻克，也许也会对人疾病有一定帮助。
感染细胞的代谢速度比寻常细胞的要快，我们将会更仔细研究感染细胞的内部结构。
我们对于病毒的特征情况尚还需要更多了解，将会继续深入研究。
这一特殊病毒我们暂取代号为R1病毒。
秘密报告，2020年8月21日
——
后人补充：
R1病毒也就是艾瑞森Flo研究所指定的PRv病毒。
